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I've been on Gleevec since September 2007 for Chronic Myelogenous Leukemia. And I can say that I didn't have any problems with iron level, but maybe I am an exception. However I know many more people who had no problems with their iron while being on Gleevec. Before it I was on another medication but it was not effective at all and had serious side effects for me. but my doctor persuaded me that it was the best possible medication in my case and it had helped many people get on lasting remission. However, it caused me to vomit from time to time, besides I had severe leg cramping and that would make me crazy. In the night I needed several blankets to calm down the cramping and fall asleep. Sometimes I spent sleepless nights and felt incredibly weak and passive next day, especially if I vomited before and had absolutely no energy to live the day out. So after 2 months I insisted on switching to another medication and he put me on Gleevec. I consider it to be a lucky decision and I am happy that this medication is so effective for me and causes no additional problems.

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Among patients who are younger than 40 years of age, who have chronic-phase CML, and who have an HLA-identical sibling donor, the leukemia-free survival rate approaches 70 percent after transplantation but is associated with a substantial cost in terms of toxicity.

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Clearly, the exact mechanisms of resistance in many patients is elusive, in part because of the relative crudeness of cytogenetic techniques in detecting all of the clinically relevant genetic lesions.

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Inhibition of the KIT signal-transduction pathway is a promising treatment for advanced gastrointestinal stromal tumors, which resist conventional chemotherapy.

The diffuse subset, characterized by the rapid development of symmetric skin thickening of proximal and distal extremities, face and trunk, is associated with visceral organ (heart, lung, kidney) involvement.

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Preoperative patients should be questioned about polyuria and polydipsia.

This will necessarily bring together professionals in genetic research (for biomarker discovery and development), patient outreach, finance (for development of pricing and economic models for stratified patient populations), and sales and marketing (for working with clinics).

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Although tumors of UGI tract are one of the common causes of UGI bleeding, majority of these tumors are primary gastrointestinal malignancies.

Phase II study of chemotherapy and stem cell transplantation for adult acute lymphoblastic leukemia or lymphoblastic lymphoma: Japan Clinical Oncology Group study 9004.

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Imatinib may decrease the serum concentration of Fludarabine.

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It is better to discuss whatever questions you have with your doctor and follow his recommendations.

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There is also controversy about whether the prognostic significance of CRLF2 abnormalities should be analyzed based on CRLF2 overexpression or on the presence of CRLF2 genomic alterations.

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COG also considers patients who are aged 13 years or older to be very high risk, although this age criterion is not utilized by other groups.

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However, drug resistance has been observed, particularly in advanced phase CML.

Efficacy results were similar in men and women and in patients younger and older than age 65.

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Phospholipase D signaling pathway, conserved biosystem Phospholipase D (PLD) is an essential enzyme responsible for the production of the lipid second messenger phosphatidic acid (PA), which is involved in fundamental cellular processes, including membra.

Drug companies that produce generic versions simply buy chemical ingredients and spend money on production, thus they can sets lower prices on their products.

Impact of Timely Switching From Imatinib to a Second-Generation Tyrosine Kinase Inhibitor After 12-Month Complete Cytogenetic Response Failure: A Chart Review Analysis.

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The cornerstone of therapy is avoidance of identifiable triggers for mast cell degranulation such as animal venoms, extremes of temperature, mechanical irritation, alcohol, or medications (e.

Impact of comorbidities on overall survival in patients with chronic myeloid leukemia: results of the randomized CML Study IV.

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The primary efficacy endpoint, rate of confirmed CCyR by 12 months, was achieved in 199 patients treated with dasatinib (76.

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Three years prior to admission, a persistently elevated total protein prompted an evaluation for multiple myeloma.

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These findings indicate that imatinib does not kill all cells in an unresected tumor and should be continued as long as disease is present (unless the patient switches to a different drug).

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Imatinib was also better tolerated than the IFN-based therapy.

Extended triple intrathecal chemotherapy trial for prevention of CNS relapse in good-risk and poor-risk patients with B-progenitor acute lymphoblastic leukemia: a Pediatric Oncology Group study.

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This could lead to a meaningful improvement in the average response rate for drugs, which is currently 50 percent across all categories and just 22 percent in oncology.

Gleevec, developed by Novartis, is a tyrosine kinase inhibitor used for the treatment of chronic myeloid leukaemia and gastrointestinal stromal tumours.

Aberrant PDGF receptors appear to deregulate the growth of a variety of cancers, such as myeloproliferative disorders, gliomas, carcinomas, melanoma, and sarcomas, including dermatofibrosarcoma protuberans.

Sunitinib offers patients with imatinib-resistant GIST a new treatment option to stop further disease progression and, in some cases, even reverse it.

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This study confirms prior observations of the efficacy of imatinib for treatment of HES and potentially further extends the indications for use of this agent for therapy of select cases of eos-CMD.

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Dasatinib provided similar complete hematologic response (87% to 92%), MCyR (61% to 63%), and CCyR (50% to 54%) rates across the four treatment arms ().

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